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This Concept Map, created with IHMC CmapTools, has information related to: Biochem A1 Neonatal Hypoglycemia and Gluconeogenesis, Neonatal pt initially presents with Macrosomia, Hypoxia leading to depressed neurological function, cortisol preventing gluconeogenesis, beta-oxidation forming NADH, liver glycogenolysis which is necessary for maitnenence of blood glucose levels, Neonatal pt initially presents with EKG, beta-oxidation forming acetyl CoA, Glucose uptake into Muscle, Neonatal pt initially presents with large amounts of adipose tissue, increased insulin:glucagon at time of birth inhibiting protein breakdown, carnitine transfers long chain fatty acids, increased insulin:glucagon at time of birth decreasing cortisol, gluconeogenesis contributing to hypoglycemia in neonate, Neonatal pt born to Mother, G-1,3-bisphosphate to G-3-phosphate, EKG showing Decreased L ventricular contractility, Macrosomia consistent w/ dx of enlarged organs, Adipose tissue contributing to hypoglycemia in neonate, glycogen stores in liver, Apgar score of 2 suggests depressed neurological function