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This Concept Map, created with IHMC CmapTools, has information related to: Tangiers, arterial macrophages are transformed into foam cells, ABCA1 found in membrane of peripheral tissues, Apo AI When combined with cholesterol, VLDL in heterozygous vs homozygous pt results in higher VLDL concentration, autosomal codominant resulting in varying expression of ABCA1, 1/2 normal HDL and normal LDL leading to decreased apoC-II, cholesterol build up in the tonsils causes phagocytosis by Tissue Macrophages, Less TAG transfer to peripheral tissue leading to Higher LDL plasma levels, apoC-II is carried by HDL, autosomal codominant phenotypes vary with heterozygotes, Tissue Macrophages form foam cells, tissue hyperplasia seen clinically as enlarged tonsils, Apo B100 is maintained on LDL, VLDL is converted to IDL, HDL when low decreased SR-B1 activity, TAGs from VLDL, foam cells build up of leads to tissue hyperplasia, familial HDL deficiency could result in Coronary artery disease, Liver by SR-B1, Apo AI stimulates activity of LCAT