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This Concept Map, created with IHMC CmapTools, has information related to: CBL A1 Diabetic Ketoacidosis, insulin:glucagon shutdown ↑ gluconeogenesis, Na+ into proximal tubule, insulin administration increases insulin:glucagon, K+ into intracellular compartment, Pt presents unconscious, beta hydroxybutyrate high level causes vomiting, hyperosmolarity decreasing ↑ plama [glucose], glucose uptake by peripheral tissue, TAG hydrolysis increasing plasma free FA, insulin:glucagon prevents shut down of ↑ gluconeogenesis, ketone bodies are acetone, type I diabetes is an autoimmune disease, beta cells in pancreas normally secrete insulin, adipose tissue stimulates TAG hydrolysis, plasma free FA leading to b-oxidation, antibodies so cannot produce & secrete insulin, ↓PaCO2 diagnostic of metabolic acidosis, glucose in urine increases H20 secretion, unconscious lab values ↓pH, insulin shifting K+